Displaying publications 1 - 20 of 949 in total

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  1. Greaves MJ
    Family Practitioner, 1978;3:27-31.
    Matched MeSH terms: Hypertension
  2. Khoo KL
    Family Practitioner, 1978;3:20-24.
    Matched MeSH terms: Hypertension
  3. Underwood T
    Family Practitioner, 1977;2(6):8-12.
    Matched MeSH terms: Hypertension
  4. Robaayah Z
    Family Practitioner, 1983;6:28-29.
    Matched MeSH terms: Hypertension
  5. Narian S
    Family Practitioner, 1978;3:38-39.
    Matched MeSH terms: Hypertension
  6. Rajakumar MK
    Family Practitioner, 1978;3:7-8.
    Matched MeSH terms: Hypertension
  7. Leong YM, Ng CW
    Family Practitioner, 1978;3:41-41.
    Matched MeSH terms: Hypertension
  8. Gill SS
    Family Practitioner, 1978;3:25-26.
    Matched MeSH terms: Hypertension
  9. Ong HT
    Med J Malaysia, 2002 Dec;57(4):510-4.
    PMID: 12733181
    Matched MeSH terms: Hypertension/diagnosis*; Hypertension/physiopathology; Hypertension/therapy*
  10. Ong HC
    Family Practitioner, 1976;2:25-30.
    Matched MeSH terms: Hypertension
  11. Sivalingam N
    Family Practitioner, 1985;8:21-24.
    Matched MeSH terms: Hypertension
  12. Balasundaram R
    Family Practitioner, 1978;3(2):14-19.
    Matched MeSH terms: Hypertension
  13. Catterall RA
    Family Practitioner, 1978;3(2):10-13.
    Matched MeSH terms: Hypertension
  14. Hoshide S, Yamamoto K, Katsurada K, Yano Y, Nishiyama A, Wang JG, et al.
    Hypertens Res, 2023 Jan;46(1):3-8.
    PMID: 36229522 DOI: 10.1038/s41440-022-00994-1
    Matched MeSH terms: Hypertension*
  15. O'Holohan DR
    Med J Malaya, 1969 Jun;23(4):260-4.
    PMID: 4242172
    Matched MeSH terms: Hypertension/diagnosis*; Hypertension/therapy*
  16. Haizal HK, Azman W, Tan KH, Choy AM, Chan CG, Amudha K, et al.
    JUMMEC, 2000;5:3-10.
    Matched MeSH terms: Hypertension
  17. Chia YC
    Med J Malaysia, 2013 Apr;68(2):101-2.
    PMID: 23629551
    Matched MeSH terms: Hypertension*
  18. Chia YC
    J Hypertens, 2016 Sep;34 Suppl 1 - ISH 2016 Abstract Book:e4-e5.
    PMID: 27753807
    Conference abstract:
    Hypertension is the leading cause of mortality worldwide. It is highly prevalent throughout the world. Even in regions liike South-East Asia (SEA) which has been perceived to be less prone to cardiovascular diseases, the prevalence of hypertension has been reported to be around 35% (1). Awareness and control of hypertension in SEA is also low, both being less than 50% each (2).Control of hypertension is an interplay between patients, doctors and system factors. One of the reasons for poor control of hypertension is resistant hypertension. Resistant hypertension is defined as blood presure that remains above goal despite being on three concurrent anti-hypertensive medications preferbaly one of which is a diuretic (3).True resistant hypertension should be differiented from secondary hypertension and pseudo-resistant hypertension. Resistant hypertension is almost always multi-factorial in aetiology. The exact prevalence of resistant hypertenion even in developed countries is not known It has been estimated that it is as high as 20-30% in clinical trial patients (4)Not many studies about resistant hypertension have been done in SEA but one done in an outpatient clinic in Thailand found it to be 7.82% Another study also done in a primary care clinc in Malaysia on 1217 patients with hypertension found the prevalence of resistant hypertension to be 8.8%. (6) Here it was found that the presence of chronic kidney disease was more likely to be associated with resistant hypertension (odds ratio [OR] 2.89, 95% confidence interval [CI] 1.56-5.35). Other factors like increasing age, female gender, presence of diabetes, obesity and left ventricular hypertrophyage which have been found to be predictors of resistant hypertension in other studies in the west were not seen in this study. There are various reasons for these findingsBut whatever the factors are that are associated with uncontrolled hypertension, the task is to sort out true resistant hypertension from pseudo-resistant hypertension and secondary casues of hypertension which may be treatable. A concerted effort is needed to reduce the BP in resistant hypertension. Failure to do so would mean a substantal increase in CV risk for the patient.
    Matched MeSH terms: Hypertension*
  19. Wang TD, Lee CK, Chia YC, Tsoi K, Buranakitjaroen P, Chen CH, et al.
    J Clin Hypertens (Greenwich), 2021 03;23(3):481-488.
    PMID: 33314715 DOI: 10.1111/jch.14123
    The prevalence of erectile dysfunction (ED) is above 40% in both Asian and non-Asian male populations after the age of 40 years. The prevalence of ED among hypertensive patients is approximately double than that in normotensive population. Pelvic arterial insufficiency is the predominant cause of ED in men aged over 50 years. Stenosis in any segment of the iliac-pudendal-penile arterial system, which is considered an erectile-related arterial axis, could lead to ED. Pharmacotherapy with lifestyle modification is effective in alleviating sexual dysfunction, yet a substantial number of patients still develop ED. Given the established applicability of angioplasty for the entire iliac-pudendal-penile arterial system, penile duplex ultrasound, and pelvic computed tomography angiography could be considered as the routine screening tools in ED patients with poor response to phosphodiesterase-5 inhibitors. Endovascular therapy for pelvic arterial insufficiency-related ED has been shown to be a safe and effective treatment option in patients who have anatomically suitable vessels and functionally significant stenoses. Clinical improvement was achieved in over 60% of patients at one year following pelvic angioplasty in the PERFECT registry from Taiwan. A 30%-40% restenosis rate in distal internal pudendal and penile arteries remains a hurdle. Angioplasty for pelvic arterial occlusive disease could be considered as a viable approach to arteriogenic ED.
    Matched MeSH terms: Hypertension*
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