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  1. Azmi MI, Takafuji M, Plein S
    Front Med (Lausanne), 2026;13:1969565.
    PMID: 42978753 DOI: 10.3389/fmed.2026.1969565
    Cardiac involvement is an important cause of morbidity and mortality in Becker muscular dystrophy. Myocardial disease may progress despite relatively limited findings on conventional echocardiography, while cardiac magnetic resonance (CMR) provides detailed assessment of ventricular function and myocardial tissue characteristics with late gadolinium enhancement (LGE), parametric mapping and strain analysis. We report a case of 57 year-old man with genetically confirmed Becker muscular dystrophy who underwent CMR examinations 12 years apart. Follow-up imaging demonstrated progressive left ventricular remodelling, a decline in left ventricular ejection fraction and interval changes in the distribution and extent of myocardial fibrosis. This case provides a long-term within-patient comparison of Becker muscular dystrophy associated cardiomyopathy and illustrates the additional information provided by serial CMR in documenting progressive myocardial involvement over time.
  2. Samat AHA, Cassar MP, Akhtar AM, McCracken C, Ashkir ZM, Mills R, et al.
    Int J Cardiol, 2024 Nov 15;415:132415.
    PMID: 39127146 DOI: 10.1016/j.ijcard.2024.132415
    BACKGROUND: The role of ECG in ruling out myocardial complications on cardiac magnetic resonance (CMR) is unclear. We examined the clinical utility of ECG in screening for cardiac abnormalities on CMR among post-hospitalised COVID-19 patients.

    METHODS: Post-hospitalised patients (n = 212) and age, sex and comorbidity-matched controls (n = 38) underwent CMR and 12‑lead ECG in a prospective multicenter follow-up study. Participants were screened for routinely reported ECG abnormalities, including arrhythmia, conduction and R wave abnormalities and ST-T changes (excluding repolarisation intervals). Quantitative repolarisation analyses included corrected QT (QTc), corrected QT dispersion (QTc disp), corrected JT (JTc) and corrected T peak-end (cTPe) intervals.

    RESULTS: At a median of 5.6 months, patients had a higher burden of ECG abnormalities (72.2% vs controls 42.1%, p = 0.001) and lower LVEF but a comparable cumulative burden of CMR abnormalities than controls. Patients with CMR abnormalities had more ECG abnormalities and longer repolarisation intervals than those with normal CMR and controls (82% vs 69% vs 42%, p 

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