Fish bone is the commonest cause of foreign body disease in Asian population. Esophageal perforation following fish bone foreign body accounts for 1-4% of the total reported cases. A 60-year-old lady presented with progressively worsening dysphagia and odynophagia over five days period following a fish meal. She sought treatment at two general practitioner clinic and a private hospital before being referred to Ear, Nose and Throat specialist. Repeated neck X-ray revealed a significant increase in prevertebral soft tissue thickness with large air-pocket tracking, small opacity at the prevertebral C3 level and subcutaneous emphysema anterior to the neck suggestive of retropharyngeal hematoma or abscess. A CT neck and thorax showed a 2.1 cm linear dense structure at the level of C7/T1 that appeared to protrude outside the esophagus in between the tracheoesophageal space. Direct laryngoscopy and repeat emergency esophagoscopy revealed a perforation at the right side of esophagus distal to cricopharyngeus with pus discharge upon milking of posterior lateral wall and a fish bone measuring 3.0 x 0.5 cm was removed from posterior wall of esophagus 17 cm from incisor. Gastrograffin study on day 10 was normal and was discharged on day 11 with Ryle’s tube feeding and to complete oral antibiotic. Fiber optic endoscopic evaluations of swallowing at two weeks follow up was normal. Subsequent review in the clinic showed full recovery without sequelae. Migrating fish bone can lead to esophagus penetration with serious complications. Mortality and morbidity from fish bone foreign body can be minimized with early diagnosis, referral and removal.
Hyperdensity of basal ganglia in computed tomography (CT) of brain is always recognised as hemorrhagic stroke or calcification. Features of hyperglycemia include muscle weakness, hypotonia, pyramidal tract signs and hemichorea-hemiballismus (HC-HB), which mimic the symptoms of stroke. Hyperdensity of the basal ganglia was reported in patient with non-ketotic hyperglycemia (NKH). Inability to recognize the hyperdensity in CT brain as a feature of NKH may lead to failure of treatment. Early recognition and reversal of hyperglycemia will improve the outcome. This was a case of an atypical presentation of stroke-like symptoms with the neuroimaging finding showing asymmetric hyperdensity of basal ganglia. Neurological examination failed to elicit any sign of HC-HB. Laboratory test showed hyperglycemia with absence of acidosis and ketonaemia. The patient was diagnosed having basal ganglia hemorrhage and referred to the Neurosurgical team. However, the stroke-like symptoms completely resolved following the normalization of glucose level. This is the only other reported case of NKH with typical neuroimaging features not associated with HC-HB. One should be aware of the possibility of NKH in the absence of movement disorder with asymmetric basal ganglia hyperdensity in CT brain. Misinterpretation as acute intracranial bleeding may result in suboptimal management of the true underlying cause.
Pain is one of commonest presentations at Emergency Department (ED). Previous studies showed inadequate pain control in ED. However, few have addressed specific, practical methods of improving the timeliness and frequency of pain control in emergency setting. This study was a randomized controlled trial in a simulated environment of an actual functioning ED using a timer device to remind care personnel to assess pain and provide analgesia at set intervals versus a “standard therapy” group without visual/audio aids. The mean documentation performance scores between timer and control groups were 94.45% + 5.85 vs 72.22% + 17.57 (p