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  1. Azmin S, Sahathevan R, Suehazlyn Z, Law ZK, Rabani R, Nafisah WY, et al.
    BMC Infect Dis, 2013;13:179.
    PMID: 23594500 DOI: 10.1186/1471-2334-13-179
    BACKGROUND: Dengue is a common illness in the tropics. Equally common are neurological complications that stem from dengue infection. However, to date, parkinsonism following dengue has not been reported in medical literature.
    CASE PRESENTATION: A previously well 18-year old man developed parkinsonism, in addition to other neurological symptoms following serologically confirmed dengue fever. Alternative etiologies were excluded by way of imaging and blood investigations.
    CONCLUSIONS: The authors detail the first reported case of parkinsonism complicating dengue fever. Keeping rare presentations of common illnesses in mind, it behoves clinicians to consider parkinsonism as a complication following dengue infection. This would prevent injudicious treatment with L-dopa and dopamine agonists. Immunosuppression with steroids has been shown to be helpful in certain cases.
    Matched MeSH terms: Parkinsonian Disorders/diagnosis*
  2. Fong CY, Hlaing CS, Tay CG, Ong LC
    Pediatr Infect Dis J, 2014 Oct;33(10):1092-4.
    PMID: 24776518 DOI: 10.1097/INF.0000000000000382
    Parkinsonism as a neurologic manifestation of dengue infection is rare with only 1 reported case in an adult patient. We report a case of a 6-year-old child with self-limiting post-dengue encephalopathy and Parkinsonism. This is the first reported pediatric case of post-dengue Parkinsonism and expands the neurologic manifestations associated with dengue infection in children. Clinicians should consider the possibility of post-dengue Parkinsonism in children with a history of pyrexia from endemic areas of dengue.
    Matched MeSH terms: Parkinsonian Disorders/diagnosis*
  3. Abdul Halim S, Mohd Amin NA
    BMJ Case Rep, 2018 Oct 21;2018.
    PMID: 30344146 DOI: 10.1136/bcr-2018-225751
    Osmotic demyelination syndrome commonly affects the pons and infrequently involves the extrapontine region. We report a patient with severe hyponatraemia who developed osmotic demyelination syndrome as a consequence of rapid sodium correction. The condition manifested as acute severe parkinsonism, bilateral ptosis and gaze impairment. MRI revealed typical features of central pontine and extrapontine myelinolysis. The patient improved gradually after treatment with a combination of levodopa, intravenous immunoglobulin and dexamethasone. However, it is important to emphasise that the improvement of neurological symptoms is not necessarily causal with these experimental therapies.
    Matched MeSH terms: Parkinsonian Disorders/diagnosis*
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