Apoptosis transcriptional mechanism of feline infectious peritonitis virus infected cells

Shuid AN 1 , Safi N 1 , Haghani A 1 , Mehrbod P 1 , Haron MS 1 , Tan SW 1 Show all authors , Omar AR 2

Affiliations 

  • 1 Institute of Bioscience, Universiti Putra Malaysia, 43400, Serdang, Selangor, Malaysia
  • 2 Institute of Bioscience, Universiti Putra Malaysia, 43400, Serdang, Selangor, Malaysia. aro@upm.edu.my
Apoptosis, 2015 Nov;20(11):1457-70.
PMID: 26386572 DOI: 10.1007/s10495-015-1172-7

Abstract

Apoptosis has been postulated to play an important role during feline infectious peritonitis virus (FIPV) infection; however, its mechanism is not well characterized. This study is focused on apoptosis and transcriptional profiling of FIPV-infected cells following in vitro infection of CRFK cells with FIPV 79-1146 WSU. Flow cytometry was used to determine mode of cell death in first 42 h post infection (hpi). FIPV infected cells underwent early apoptosis at 9 hpi (p 

* Title and MeSH Headings from MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.

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