Displaying publications 1 - 20 of 30 in total

  1. Hamizan AW, Yean KT, Abdullah A
    Int J Pediatr Otorhinolaryngol, 2012 Mar;76(3):455-9.
    PMID: 22281372 DOI: 10.1016/j.ijporl.2012.01.002
    A 3-year-old child presented with congenital bilateral facial nerve palsy with bilateral profound sensorineural hearing loss. High Resolution Computed Tomogram (HRCT) of the temporal bones found bilateral atresia of cochlear nerve canals, incomplete partition of the cochleae and narrow facial nerve canals. Magnetic resonance imaging (MRI) revealed bilateral hypoplasia of facial nerves and aplasia of both vestibulocochlear nerves. There have been no other reported cases with this presentation. The possible aetiology and treatment options for the patient are discussed. We highlighted the review of aplasia/hypoplasia of the facial nerve and hypoplasia of cochlear nerve canal.
    Matched MeSH terms: Facial Paralysis/complications*; Facial Paralysis/congenital*; Facial Paralysis/diagnosis
  2. Asma A, Marina MB, Mazita A, Fadzilah I, Mazlina S, Saim L
    Singapore Med J, 2009 Dec;50(12):1154-7.
    PMID: 20087551
    This study aims to review the management and discuss the outcome of patients with iatrogenic facial nerve palsy.
    Matched MeSH terms: Facial Paralysis/etiology*; Facial Paralysis/radiography; Facial Paralysis/therapy*
  3. Tan CY, Yuki N, Shahrizaila N
    J Neurol Sci, 2015 Nov 15;358(1-2):409-12.
    PMID: 26277343 DOI: 10.1016/j.jns.2015.08.009
    Miller Fisher syndrome is characterised by the triad of ophthalmoplegia, ataxia and areflexia. However, facial palsy can occur during the course of the illness although development of facial palsy when other cardinal signs of Miller Fisher syndrome have reached nadir or improving, is unusual. This delayed appearance of facial palsy can be easily overlooked by the treating clinician. Here, we report four patients with Miller Fisher syndrome and delayed-onset facial palsy. We discuss the possible underlying reasons behind the delay in facial palsy.
    Matched MeSH terms: Facial Paralysis
  4. Badaruddin A, Choo MM
    Malays Fam Physician, 2021 Mar 25;16(1):117-120.
    PMID: 33948150 DOI: 10.51866/cr1108
    Otitis externa is an infection of the external auditory canal. It rarely results in facial palsy except in severe cases such as necrotizing otitis externa, which is a life-threatening invasive infection of the external auditory canal. Early recognition with prompt and appropriate treatment of necrotizing otitis externa is crucial to prevent more sinister complications. Here we report a case of an elderly gentleman who presented with otitis externa and developed facial palsy a month later. We identified possible problems that may have led to the complication so that such an occurrence can be prevented in the future.
    Matched MeSH terms: Facial Paralysis
  5. Rajinder S, Nik Adilah NO
    Malays Fam Physician, 2017;12(3):25-27.
    PMID: 29527276 MyJurnal
    The presence of a tick in the ear is an uncommon problem encountered by the department of otorhinolaryngology. A tick infestation in the ear can be a traumatising experience for the patient. Here, we report a case of a woman who presented with left facial weakness due to the presence of a tick in the external auditory canal.
    Matched MeSH terms: Facial Paralysis
  6. Long YT, bin Sabir Husin Athar PP, Mahmud R, Saim L
    Asian J Surg, 2004 Jul;27(3):176-9.
    PMID: 15564156
    A 6-year review of complications of mastoid surgery between June 1995 and June 2001 revealed five cases with serious iatrogenic complications from mastoid surgery, of which four were facial nerve palsy and two were labyrinthine fistula. One of these patients had concomitant facial nerve palsy and labyrinthine fistula. There were two cases of complete facial nerve palsy (House Brackmann grade VI) and two cases of incomplete palsy (House Brackmann grades IV and V). The second genu was the site of injury in three of the four cases. Of the four cases with facial nerve palsy, two patients had full recovery (House Brackmann grade I), one recovered only to House Brackmann grade III, and one was lost to follow-up. Both patients with labyrinthine fistula had postoperative vertigo and profound sensorineural hearing loss. The site of iatrogenic fenestration was the lateral semicircular canal in both cases.
    Matched MeSH terms: Facial Paralysis/etiology*; Facial Paralysis/surgery
  7. Yeoh TL, Mahmud R, Saim L
    Med J Malaysia, 2003 Aug;58(3):432-6.
    PMID: 14750385
    A four years review from June 1998 to June 2002 of traumatic facial nerve paralysis from temporal bone fractures that required surgical intervention is presented. The aim of this clinical presentation was to determine the current pattern of cases with traumatic facial paralysis which required surgical intervention at our center. There were six cases, of which four (66%) were longitudinal fractures, one each (17%) had transverse fracture and fracture over the lateral wall of mastoid. Hearing loss (83%) was the commonest associated clinical symptom. All cases underwent decompression via the transmastoid surgical approach. Intraoperative findings revealed oedema of facial nerve involving vertical segment and horizontal segment in three cases each respectively. Two cases had concomitant bony impingement. The facial nerve functions in four cases (66%) and one case recovered to House Brackmann grade 2 and 4, 12 months and 3 months respectively postsurgery. The case with transverse fracture remained as House Brackmann grade 5 after two years.
    Matched MeSH terms: Facial Paralysis/etiology*; Facial Paralysis/surgery*
  8. Selvamalar V, Othman NAN, Daud MK
    Acta Medica (Hradec Kralove), 2021;64(1):36-41.
    PMID: 33855957 DOI: 10.14712/18059694.2021.6
    Malignant otitis externa is an inflammation of the external auditory canal with preceding osteomyelitis of the temporal bone and the adjacent structures that could be potentially lethal. Malignant otitis externa may present with cranial nerve involvements and massive spread of disease mimicking nasopharyngeal carcinoma or any other malignancies on imaging. Two elderly patients who presented with severe otalgia and significant facial nerve palsy and lower cranial nerve palsies showing extensive spread of disease are reported in this case series. They both had resolution of disease after a prolonged course of antibiotics and cortical mastoidectomy for disease clearance in one of them.
    Matched MeSH terms: Facial Paralysis/diagnosis; Facial Paralysis/therapy
  9. Oo KT, Tay KS, Law WC, Shatriah I
    Taiwan J Ophthalmol, 2019 12 05;10(3):235-238.
    PMID: 33110759 DOI: 10.4103/tjo.tjo_69_19
    Over the past decade, the discovery of disease-specific aquaporin-4 antibodies has led to a better understanding of the diverse spectrum of disorders that are associated with neuromyelitis optica. Brainstem manifestations have been increasingly recognized in this disease. However, multiple cranial nerve palsies as an initial presentation of neuromyelitis optica are uncommon. We report a rare case of anti-aquaporin-4 antibody-positive neuromyelitis optica that presented with unilateral abducens and facial nerve palsies. Notably, this case did not involve the optic nerve or the spinal cord. Diagnosing neuromyelitis optica that presents as an isolated acute brainstem syndrome is challenging, but the outcome may be devastating if the diagnosis is delayed.
    Matched MeSH terms: Facial Paralysis
  10. Sabir Husin Athar PP, Yahya Z, Mat Baki M, Abdullah A
    Malays J Med Sci, 2009 Apr;16(2):38-9.
    PMID: 22589657
    Benign parotid neoplasm and inflammatory processes of the parotid resulting in facial paralysis are extremely rare. We report a 72-year-old Malay female with poorly-controlled diabetes mellitus who presented with a painful right parotid swelling associated with right facial nerve palsy. The paralysis (Grade VI, House and Brackmann classification) remained after six months.
    Matched MeSH terms: Facial Paralysis
  11. Azarisman, S.M.S., Shahrin, T.C.A., Marzuki, A.O., Fatnoon, N.N.A., Rathor, M.Y.
    Bilateral simultaneous facial nerve palsy is an extremely rare clinical entity and may occur in association with a variety of neurological, infectious, neoplastic or degenerative disorders. We describe a patient, who presented with facial diplegia and normal reflexes on examination. During the entire hospitalization, he developed no motor weakness and remained ambulatory. Whether treatment is warranted for this and other milder variants of Gullain-Barré syndrome is also discussed. Atypical presentations with preserved or brisk reflexes, can be a diagnostic dilemma.
    Matched MeSH terms: Facial Paralysis
  12. Baharudin, A., Din Suhaimi, S., Omar, E.
    Schwannomas are benign slow growing lesions arising from the Schwann cells that ensheath the axons of the peripheral, cranial and autonomic nervous systems. Intracranial schwannomas develop from the facial nerve much more rarely than from the vestibular or trigeminal nerves. Ancient schwannoma is an unusual histological variant of this rare disease. A 48 years old man who had recurrent facial nerve paralysis and right external auditory mass is presented in this case report.
    Matched MeSH terms: Facial Paralysis
  13. Mazita, A., Zahirrudin, Z., Saim, L., Asma, A.
    Medicine & Health, 2010;5(2):86-92.
    Facial nerve schwannoma is a rare slow growing benign tumour which arises from the Schwann cell of the neurilemma. A retrospective review of 6 patients who had been diagnosed with facial nerve schwannoma between 1998 and 2008 was conducted. There was equal distribution of male and female patients. The mean age was 42 years (range 19 to 66 years). The tumour originated in the internal auditory canal (2 patients), intra-temporal (3 patients) and intraparotid (1 patient) segments of the facial nerve. All tumours were successfully removed and facial nerve continuity was pre-served in 2 cases. The presenting symptoms of facial nerve schwannoma are non specific and dependent on the site of tumour origin. It is a great mimicker of other lesions that can present at the same location. The surgeon should have a high index of suspicion when patients present with progressive facial nerve palsy. Patients should always be counselled regarding risk of facial paralysis because the diagnosis of facial nerve schwannoma is often confirmed intra-operatively.
    Matched MeSH terms: Facial Paralysis
  14. Zamzil Amin, A., Baharudin, A., Shahid, H., Din Suhaimi, S., Nor Affendie, M.J.
    A tick in the ear is a very painful condition and removal is difficult because it grips firmly to the external auditory canal or tympanic membrane. Facial paralysis is a rarely reported localised neurological complication of an intra-aural tick infestation. The pathophysiology of localised paralysis is discussed, together with the safe way of handling patients with an intra-aural tick infestation.
    Matched MeSH terms: Facial Paralysis
  15. Noorizan Y, Chew YK, Khir A, Brito-Mutunayagam S
    Med J Malaysia, 2009 Jun;64(2):172-3.
    PMID: 20058583 MyJurnal
    Facial nerve palsy with a parotid mass is usually associated with malignant neoplasm of parotid gland. Its occurrence as a complication of parotid abscess is extremely rare. A literature review revealed only 16 cases of facial nerve palsy associated with suppurative parotitis or parotid abscess were reported. We present a case of deep parotid abscess which is complicated by facial nerve dysfunction. Underlying neoplasia was excluded.
    Matched MeSH terms: Facial Paralysis/etiology*
  16. Saniasiaya J, Nik Othman NA, Mohamad Pakarul Razy NH
    Braz J Otorhinolaryngol, 2016 05 24;86(1):130-132.
    PMID: 27269254 DOI: 10.1016/j.bjorl.2016.04.012
    Matched MeSH terms: Facial Paralysis/etiology
  17. Jalaludin MA
    Methods Find Exp Clin Pharmacol, 1995 Oct;17(8):539-44.
    PMID: 8749227
    Sixty patients with Bell's palsy were included in an open randomized trial. Patients were assigned into three treatment groups: steroid (group 1), methylcobalamin (group 2) and methylcobalamin + steroid (group 3). Comparison between the three groups was based on the number of days needed to attain full recovery, facial nerve scores, and improvement of concomitant symptoms. The time required for complete recovery of facial nerve function was significantly shorter ( p < 0.001) in the methylcobalamin (mean of 1.95 +/- 0.51 weeks) and methylcobalamin plus steroid groups (mean of 2.05 +/- 1.23 weeks) than in the steroid group (mean of 9.60 +/- 7.79 weeks). The facial nerve score after 1-3 weeks of treatment was significantly more severe (p < 0.001) in the steroid group compared to the methylcobalamin and methylcobalamin plus steroid groups. The improvement of concomitant symptoms was better in the methylcobalamin treated groups than the group treated with steroid alone.
    Matched MeSH terms: Facial Paralysis/drug therapy*
  18. Khanijow VK
    Med J Malaysia, 1991 Sep;46(3):259-61.
    PMID: 1667539
    A case of an adenoid cystic carcinoma of the minor salivary glands of then nasal cavity is reported. The tumour had spread locally and by perineural spread to the internal auditory mentus, causing facial nerve palsy.
    Matched MeSH terms: Facial Paralysis/etiology*
  19. Siang PG, Ying XT, Dayang Suhana AM, Ing PT
    Med J Malaysia, 2020 05;75(3):281-285.
    PMID: 32467545
    INTRODUCTIONS: Facial nerve palsy (FNP) occurs in 7-10% of temporal bone fractures. The aim of this study was to review the surgical outcome of nine patients with severe to complete traumatic facial nerve (FN) injury.

    METHODS: The patients were evaluated clinically and FNP was graded using the House Brackmann (HB) scale. High resolution computerized tomography (HRCT) of the temporal bone was used to evaluate temporal bone fractures. Transmastoid facial nerve decompression was performed and the facial nerve function was re-evaluated in subsequent follow ups.

    RESULTS: There were five cases with immediate onset and four with delayed onset of FNP. Only three cases had pure temporal bone fractures, the others were associated with other life threatening injuries. The sensitivity and specificity of HRCT temporal bone to detect the obvious facial canal fracture line were 50% and 40% respectively. 75% of patients with immediate onset of HB grade VI FN palsy who were operated within a month recovered completely. Surgeries for the delayed onset FNP were performed at a mean of 70 days (range 51-94). All recovered to HB grade II-III from severe FNP.

    CONCLUSIONS: Our study demonstrated that transmastoid FN decompression surgery was beneficial to traumatic nerve injury. Early intervention resulted in better outcomes. However, FN function could still be salvaged even in delayed FN decompression.

    Matched MeSH terms: Facial Paralysis/surgery*
  20. Ngow HA, Wan Khairina WM, Hamidon BB
    Singapore Med J, 2008 Oct;49(10):e278-80.
    PMID: 18946598
    Bell's palsy is a benign lower motor neuron facial nerve disorder. It is almost always unilateral. We report a 20-year-old nulliparous woman with five episodes of recurrent Bell's palsy. A review of recent medical literature revealed a paucity of case reports involving an individual with five episodes of recurrent Bell's palsy, with none found in Asian neurology medical literature. Despite the multiple episodes of Bell's palsy recurrences, the patient did not suffer much neurological sequelae from the disease.
    Matched MeSH terms: Facial Paralysis/complications
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